Hematopoietic and Cardiac GPR55 Synchronize Post-Myocardial Infarction Remodeling

Scientists investigated the role of GPR55 in cardiac physiology and post-myocardial infarction (MI) inflammation and remodeling. Global GPR55 deficiency mitigated MI-induced fetal gene re-programming and cardiomyocyte hypertrophy, culminating in aggravated LV dilatation and infarct expansion.